Capecitabine, DNA/RNA Synthesis Inhibitor
Por um escritor misterioso
Descrição
Capecitabine is an oral proagent that is converted to its active metabolite, 5-FU, by thymidine phosphorylase. - Mechanism of Action & Protocol.
5-Fluorouracil (5-FU) resistance and the new strategy to enhance the sensitivity against cancer: Implication of DNA repair inhibition - ScienceDirect
Frontiers 5-Fluorouracil: A Narrative Review on the Role of Regulatory Mechanisms in Driving Resistance to This Chemotherapeutic Agent
DNA and RNA Synthesis: Antifolates
5-Fluorouracil
Chemotherapy and Transplantation: The Role of Immunosuppression in Malignancy and a Review of Antineoplastic Agents in Solid Organ Transplant Recipients - American Journal of Transplantation
Capecitabine, C15H22FN3O6
Targeting nucleotide metabolism as the nexus of viral infections, cancer, and the immune response
Fluoropyrimidine-induced toxicity and DPD deficiency.. A case report of early onset, lethal capecitabine-induced toxicity and mini review of the literature. Uridine triacetate: Efficacy and safety as an antidote. Is it accessible outside
Capecitabine induces hand-foot syndrome through elevated thymidine phosphorylase-mediated locoregional toxicity and GSDME-driven pyroptosis that can be relieved by tipiracil
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por adulto (o preço varia de acordo com o tamanho do grupo)
