Degradation of HMGB1 and PARP-1 in HeLa cells after infection with C.
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Descrição
Analyses of different endpoints of cellular physiology of HeLa
Persistent Chlamydia trachomatis Infection of HeLa Cells Mediates
Inhibiting the cytoplasmic location of HMGB1 reverses cisplatin
CPAF is responsible for HMGB1 and PARP-1 degradation in C
Vpr interacts with PARP-1 and inhibits its nuclear localization
The Role of PARPs in Inflammation-and Metabolic-Related Diseases
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